十二指腸潰瘍 / Duodenal Ulcer

SKU Rehab0085
HK$879.00
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十二指腸潰瘍

關於幽門螺桿菌在十二指腸潰瘍中的致病作用存在爭議。幽門螺桿菌(helicobacterpylori)是一種彎曲的革蘭氏陰性微需氧菌,存在於胃粘膜層或粘附於胃上皮層。從細菌的發現開始,它就對公眾健康具有重要意義,而且感染的流行率和嚴重程度在不同人群中差异很大。H、幽門螺桿菌是各種疾病的風險,而宿主反應的程度,如胃部炎症和壁細胞的酸分泌量影響感染的結果

綜述證據支持幽門螺桿菌感染和十二指腸潰瘍之間的强烈因果關係,因為患者更可能被隨後引起十二指腸潰瘍的毒株感染。因此,根除幽門螺桿菌感染可降低十二指腸潰瘍的發生率,並通過减少基礎胃泌素釋放和胃酸分泌而防止其復發,而不影響壁細胞敏感性。另一方面,一些研究表明,幽門螺桿菌感染與十二指腸潰瘍的發生無關,這種缺乏關聯性表明十二指腸潰瘍有不同的發病機制。儘管各種研究對幽門螺桿菌與十二指腸潰瘍的因果關係存在爭議,但因果關係標準證明幽門螺桿菌感染與十二指腸潰瘍之間存在因果關係。其他因素也與十二指腸潰瘍的發生有關,這些因素也與疾病患病率的差异有關。

十二指腸潰瘍-多學科研證復康方案

  • 藥物及其他醫療方案,請遵醫從醫生建議
  • 飲食營養請依營養師指引
  • 運動及或心理輔導,請參考各專家建議
  • 為免錯誤診斷及漏診,請上傳最近一個月醫院的專業診斷報告,如證實是此症,則可參加試用期不如意退款計劃#。

可能適合的補充劑支援:

標準組合:

  • 試用時期: 10天

# <試用期不如意退款計劃>,請參考相關文件

If you have this disease, please follow your doctor's complete medical plan. You must consult the attending doctor before using the multidisciplinary rehabilitation plan. If your attending doctor does not recommend you to join the supplement conditioning combination, please do not use it. If you need to seek second opinion from other doctors, you can contact our online "Doctors Without Borders", or another professional doctor in your own city.

Duodenal Ulcer

There are controversies on the causal role of H. pylori in duodenal ulceration. Helicobacter pylori are curved gram-negative microaerophilic bacteria found at the layer of gastric mucous or adherent to the epithelial lining of the stomach. It’s a public health significance bacteria starting from discovery, and the prevalence and severity of the infection varies considerably among populations. H. pylori are a risk for various diseases, while the extent of host response like gastric inflammation and the amount of acid secretion by parietal cells affects the outcome of infection.

The review evidence supports a strong causal relation between H. pylori infection and duodenal ulcer, as patients are more likely to be infected by virulent strains which later cause duodenal ulceration. Thus, eradication of H. pylori infection decreases the incidence of duodenal ulcers, and prevents its recurrence by reducing both basal gastrin release and acid secretion without affecting parietal cell sensitivity. On the other hand, some studies show that H. pylori infection is not associated with the development of duodenal ulcers and such a lack of association revealed that duodenal ulceration has different pathogenesis. Despite controversies observed in the causal role of H. pylori to duodenal ulceration by various studies, Hill criteria of causation proved the presence of a causal relation between H. pylori infection and duodenal ulcers. Other factors are also responsible for the development of duodenal ulcers and such factors are responsible for the differences in the prevalence of the diseases.

Multidisciplinary research and rehabilitation program for Duodenal Ulcer

  • Please follow the doctor's advice for drugs and other medical plans
  • Please follow the guidance of nutritionist
  • For sports or psychological counseling, please refer to the experts' suggestions
  • In order to avoid misdiagnosis and missed diagnosis, please upload the professional diagnosis report of the hospital in the latest month. If it is confirmed to be this disease, you can participate in the .#

Possible supplements supporting:

Standard combination:

Trial period: 10 days

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