尿酸關節痛 | Uric Acid Arthralgia

尿酸關節痛

痛風是一種全身性疾病，由尿酸單鈉晶體（MSU）沉積在組織中引起。血清尿酸（SUA）高於特定閾值是形成尿酸晶體的必要條件。儘管高尿酸血症是痛風的主要致病缺陷，但許多高尿酸血症患者並沒有發展成痛風，甚至沒有形成尿酸結晶。事實上，只有5%的高尿酸血症超過9毫克/分昇的人會患痛風。因此，有人認為遺傳傾向等其他因素也參與了痛風的發病。

尿酸單鈉晶體可以沉積在所有組織中，主要是在形成tophi的關節及其周圍。痛風的診斷主要是通過關節液體抽吸或tophi抽吸來鑒別病理性MSU晶體。痛風的早期表現是急性關節炎症，可通過非甾體抗炎藥或秋水仙堿迅速緩解。腎結石和腎結石是晚期表現。通過飲食調整和使用降低血尿酸的藥物來降低SUA水准低於沉積閾值是痛風治療的主要目標。這導致MSU晶體溶解，防止進一步攻擊。

Ref: https://www.ncbi.nlm.nih.gov/pmc/articles/PMC55121...

尿酸關節痛- 多學科研證復康方案

• 藥物及其他醫療方案,請遵醫從醫生建議
• 飲食營養請依營養師指引
• 運動及或心理輔導,請參考各專家建議

• 為免錯誤診斷及漏診,請上傳最近一個月醫院的專業診斷報告,如證實是此症,則可參加試用期不如意退款計劃#。

可能適合的補充劑支援:

標準組合:

1. 結晶研究 X1
2. 結晶研究霜劑 X1

• 試用時期: 1星期

# <試用期不如意退款計劃>,請參考相關文件

本產品並非根據《藥劑業及毒藥條例》或《中醫藥條例》注册。任何對其提出的索賠均不受此類登記評估的約束。本產品不用於診斷、治療或預防任何疾病。

If you have this disease, please follow your doctor's complete medical plan. You must consult the attending doctor before using the multidisciplinary rehabilitation plan. If your attending doctor does not recommend you to join the supplement conditioning combination, please do not use it. If you need to seek second opinion from other doctors, you can contact our online "Doctors Without Borders", or another professional doctor in your own city.

Uric Acid Arthralgia

Gout is a systemic disease that results from the deposition of monosodium urate crystals (MSU) in tissues. Increased serum uric acid (SUA) above a specific threshold is a requirement for the formation of uric acid crystals. Despite the fact that hyperuricemia is the main pathogenic defect in gout, many people with hyperuricemia do not develop gout or even form UA crystals. In fact, only 5% of people with hyperuriceamia above 9 mg/dL develop gout. Accordingly, it is thought that other factors such as genetic predisposition share in the incidence of gout.

Monosodium urate crystals crystals can be deposited in all tissues mainly in and around the joints forming tophi. Gout is mainly diagnosed by identification of the pathognomonic MSU crystals by joint fluid aspiration or in tophi aspirate. Early presentation of gout is an acute joint inflammation that is quickly relieved by NSAIDs or colchicine. Renal stones and tophi are late presentations. Lowering SUA levels below deposition threshold either by dietary modification and using serum uric acid lowering drugs is the main goal in management of gout. This results in dissolution of MSU crystals preventing further attack.

Ref: https://www.ncbi.nlm.nih.gov/pmc/articles/PMC55121...

Multidisciplinary research and rehabilitation program for Uric Acid Arthralgia

• Please follow the doctor's advice for drugs and other medical plans
• Please follow the guidance of nutritionist
• For sports or psychological counseling, please refer to the experts' suggestions
• In order to avoid misdiagnosis and missed diagnosis, please upload the professional diagnosis report of the hospital in the latest month. If it is confirmed to be this disease, you can participate in the .#

Possible supplements supporting:

Standard combination:

• Crystallized Study X1
• Crystallized Study Cream X1

Trial period: 1 weeks